The etiology of prostatitis is currently unclear; its formation/development can be attributed to many factors:
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infection (microcolonies, or biofilms on the surface of prostate stones),
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immunopathology diseases,
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stress, for which we mean the increased tone of the muscles in/of the pelvis and the increased sensitivity of the mechanoreceptors in the muscle fascia
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hormonal factors
Most frequently, it mutates in an ascending way, when pathogens primarily settle in the lower part of the urethra, and then, due to various factors, move up and enter the prostate. With regards to colonisation, sexual contact plays the most habitual role. In such cases, firstly the vagina becomes colonised or infected with the bacteria and then during sexual contact, pathogens can temporarily colonise the male uretha. After this, depending on the virulence of the bacteria and the body’s immune system, prostatitis can develop.
Lately, increased research into the disease from an immunological perspective has revealed the possibility of an autoimmune origin.
In addition to this, there is a bigger emphasis on existing urinary dysfunction, the background for which is based on :
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the abnormally increased closing pressure of the external sphincter
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the incomplete relaxation of the bladder,
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the increased intraprostatic pressure causing urine reflux to develop in the prostateducts and the acinars.
In developing prostatitis, neither the haematogenous nor the lymphogenous routes have any real influence
